addition to employee's left eye condition, Dr. Krummenacher was concerned about the immediate threat to vision in the right eye. On September 7, 2004, Dr. Krummenacher found no change in the vitreous hemorrhage which overlaid a tractional retinal detachment in employee's left eye. Dr. Krummenacher believed the situation in employee's left eye was dire and potentially inoperable, and referred employee back to Dr. Phillips to get employee's diabetes under control in anticipation of surgery. On October 4, 2004, Dr. Krummenacher performed a lensectomy, vitrectomy, membranectomy, fluid-gas exchange, endophotocoagulation, and indirect laser photocoagulation of the left eye, and laser surgery on the progressive retinopathy in the right eye. Dr. Krummenacher's post-operative diagnosis was proliferative diabetic retinopathy of both eyes. Employee's vision continued to deteriorate over the course of several subsequent visits with Dr. Krummenacher. On December 13, 2004, Dr. Krummenacher again performed surgery on both eyes; his post-operative diagnoses were proliferative diabetic retinopathy of both eyes, with tractional retinal detachment and vitreous hemorrhage in the left eye.
Dr. Phillips examined employee again on March 30, 2005. During the exam, employee discussed disability benefits with the doctor and whether employee's condition was job related. At this visit, employee reported that he felt something pop in his eye in late August 2004.
Dr. Krummenacher saw employee again on April 28, 2005. Employee's right eye was improving, but the left eye was irreparable with no light perception due to poor retinal circulation. Dr. Krummenacher saw no evidence that an injury in August 2004 affected employee's long term vision. Dr. Krummenacher opined that employee has a classic case of advanced proliferative diabetic retinopathy, and this, and only this, is the cause of employee's permanent disability. Dr. Krummenacher believed employee was at serious risk of further vision loss from poor circulation, and noted that employee was urged to do his utmost to control his underlying diabetes as aggressively as possible.
Employee sought treatment for left eye pain, headaches, dizziness, and facial pressure and pain from September 2005 through February 2007. Treating doctors consistently diagnosed high blood pressure and diabetes.
Dr. Joan Pernoud examined employee on June 7, 2005. Dr. Pernoud found employee's visual acuity to be 20/100 in the right eye and only peripheral light perception in the left eye. Dr. Pernoud found that employee had severe loss of vision in his left eye and that the left optic nerve was essentially dead. Employee also had definite signs of diabetic retinopathy in his right eye. Dr. Pernoud opined that employee suffered from severe ischemia, or lack of proper blood supply, to his eyes with the left being more severe. Dr. Pernoud opined that employee's acute left eye condition was caused by employee's performance of a Valsalva maneuver when he pulled a salt pan from under a railcar at work on August 28, 2004. Dr. Pernoud explained that a Valsalva maneuver occurs when someone takes a deep breath and holds it forcefully in preparation for heavy lifting or pulling a heavy object. Dr. Pernoud explained further that a Valsalva maneuver results in increased pressure to the veins coming from the head, so that blood does not drain properly from the head.
Dr. Pernoud outlined four different possibilities that could have occurred as a result of employee's performance of the Valsalva maneuver. First, pressure from engorged retinal veins could have compressed the adjacent arteries, which, being narrow and weakened from thirty-seven years of diabetes, could have occluded, causing permanent vision loss; this could have happened absent any trauma or Valsalva maneuver. Second, if employee had carotid artery disease, ischemic ocular syndrome could have occurred, causing the symptoms of a red painful eye, with a fixed dilated pupil, and a cataract. Third, abnormal vessels in the iris could have bled into the anterior chamber of the eye, causing glaucoma, corneal edema, and a longstanding increase in intraocular pressure to the point of destroying the optic nerve fibers; this also could have occurred spontaneously. Fourth, Valsalva retinopathy could have resulted from intraocular vitreous bleeding, as employee's fragile venous system ruptured from vascular engorgement. Dr. Pernoud believed that the fourth possibility was more consistent with employee's history, and probably best reflects what occurred.
In Dr. Pernoud's opinion, employee's performance of the Valsalva maneuver while engaged in his work duties was the precipitating or triggering event, and was the substantial factor in causing employee's left eye condition. Dr. Pernoud opined that diabetic retinopathy was the cause of employee's right eye condition. Dr. Pernoud acknowledged that employee had diabetic retinopathy before any reported injury based on the notes from employee's September 1999 examination by Dr. Phillips. Dr. Pernoud also acknowledged that employee's underlying condition is diabetic retinopathy in both eyes, and that this condition is fairly severe because employee has had diabetes for such a long period of time, and because employee did not follow up as he probably should have regarding care and treatment of his diabetes. Dr. Pernoud acknowledged that employee's underlying diabetic condition was so severe that his left eye complaints could have occurred spontaneously, absent any traumatic event or Valsalva maneuver.
Dr. Elliot Korn examined employee on January 5, 2006, and October 4, 2007. On January 5, 2006, Dr. Korn found employee's right eye visual acuity to be 20/50 minus three. There was no light perception in the left eye. Pathology in employee's left eye included a swollen cornea, scarring, and new blood vessel formation of the iris secondary to a period of severe ischemia. Dr. Korn diagnosed a blind left eye, severe ischemic and proliferative diabetic retinopathy of the left eye, and iris revascularization of the left eye. Dr. Korn opined that neither employee's action of pulling the salt pan on August 28, 2004, nor the performance of a Valsalva maneuver, were substantial causative factors of employee's condition. Dr. Korn acknowledged that a Valsalva maneuver can cause hemorrhages in the eye, but explained that if there are other key factors present, a Valsalva maneuver would not be a substantial factor. Dr. Korn opined that there are many other factors present in employee's case that better explain the pathology of his left eye, namely, employee's preexisting diabetic retinopathy, longstanding poor glucose control, and malignant hypertension. Dr. Korn agreed with Dr. Krummenacher that employee's condition was a classic case of advanced proliferative diabetic retinopathy. Dr. Korn noted that when employee was treated at the Christian Hospital emergency room on August 28, 2004, his blood pressure was extremely elevated; Dr. Korn explained that blood pressure that high can cause retinal
hemorrhages and blindness. Dr. Korn believed that the treatment employee received was reasonable and necessary to address employee's diabetic retinopathy, but that the treatment was not related to the August 2004 incident. Dr. Korn believed that any lost time in connection with treatment was not due to the August 2004 incident.
In Dr. Korn's opinion, even if employee performed a Valsalva maneuver, it would not be a substantial causative factor in the ultimate diagnosis or condition of employee's left eye. Dr. Korn explained that a Valsalva maneuver, by definition, is self-limited with low possibility of causing symptomatic pathology. Ultimately, Dr. Korn opined that the work incident on August 28, 2004, was not a substantial factor causing employee's condition, diagnosis, or need for treatment. Rather, in Dr. Korn's opinion, employee's poor diabetic compliance caused severe secondary malignant hypertension and diabetic retinopathy with loss of vision in the left eye, and very guarded vision in the right eye.