In January, the employee's anxiety was better. Dr. Chaudhari noted the employee's right upper brachial plexus injury had an excellent result but there was right deltoid atrophy. The employee had cervical spondylosis; anxiety and panic reactions; left carpal tunnel syndrome;
work related trauma with traction and neuropraxia of both median nerves and musculoskeletal injury involving wrist and forearm bilaterally; resolved muscle stiffness in the left paraspinal and cervical regions; C6 radiculopathy bilaterally which has resolved to a great extent; and moderate supraspinatus and deltoid atrophy on the right. Dr. Chaudhari increased the Xanax and added Effexor.
2000:
In February, the employee continued to have a few panic attacks and Xanax was continued. In November, Dr. Chaudhari prescribed Xanax.
The employee testified that he thought he settled his 1994 Illinois workers' compensation case for 6 %. He could not think of any activities that he gave up due to the neck injury. He became a foreman but was never told that he was made a foreman to compensate for his 1987 or 1994 injuries. Prior to March of 2001, he regained considerable use of his right arm and was able to go back to plumbing and pipe fitting. He had to adjust how he worked and he had to do everything left side dominant but was able to use his right arm to stabilize things. The pain improved but after a hard day his arm pain increased. His right arm range of motion was limited and he had loss of strength. He developed carpal tunnel syndrome in his left hand from using it so much to compensate for his right arm deficits. He continued to have rib pain and neck pain. Working aggravated his left hand, right arm, shoulder, and his ribs. He had panic attacks. Overhead work aggravated his neck and shoulder. Power tools aggravated his carpal tunnel syndrome. Prior to 2001 he was taking medications for anxiety.
2001:
The employee testified that he had been working at G.A. Rich and Sons for about a year prior to March 16, 2001. On March 16, 2001, he was working as a foreman, slipped on a muddy board, fell backwards, landed on his tailbone, and hit his head. He injured his neck and low back.
On March 26, the employee saw Dr. Ritter. The employee had been on Xanax for panic attacks on an as needed basis. He had a significant injury when he was crushed in a trench in 1987. Dr. Ritter diagnosed a lumbar spine strain/sprain, and prescribed Motrin and physical therapy. The employee had therapy for his neck and low back from March 28 through April 13.
On April 11, Dr. Gardner noted that the employee had atrophy of the anterior deltoid and pectoralis muscles on the right side with weakness. He had diffuse decrease in pin sensation in the right arm; and an area of decreased sensation along the left wrist in the approximate distribution of C7. Dr. Gardner recommended an MRI of the cervical and lumbar spine and an EMG and nerve conduction study of the left arm.
The employee had MRIs on April 20, 2001 for post traumatic neck and low back pain. The lumbar MRI showed a disc herniation at L5-S1 that could be impinging upon a nerve root with smaller disc herniations at L3-4 and L4-5 and associated degenerative changes. The
cervical MRI was markedly abnormal and demonstrated severe spinal cord compression with secondary edema at the C4 and C5 vertebral levels caused by disc herniations at C4-5 and C5-6; and a smaller disc herniation with annular tear at C6-7.
The April 22 myelogram showed an extradural defect at L3-4 with bilateral nerve root compression of L4. There was extradural defect at C4-5 through C6-7 with associated attenuated nerve roots bilaterally at C5-6 and C6-7. Dr. Gardner stated that the cervical MRI showed severe spinal cord compression with edema at C4 and C5 caused by disc herniations and degenerative changes. There was a milder disc herniation at C6-7. The lumbar MRI showed diffuse degenerative disc disease along with disc herniation extending to the right neural foramina at L5S1. The April 23 CT scan showed a disc bulge at L3-4 which caused mild flattening of the thecal sac that extended into the inferior aspect of the left neural foramen. A left-sided herniated disc at L4-5 caused moderate narrowing of the neural foramen. There was a mild disc bulge at L5-S1 with narrowing of the neural foramen on the right secondary to degenerative facet joint disease. The post myelogram CT scan showed marked disc space narrowing at C6-7 with moderate narrowing at C4-5 and C5-6 with hypertrophic spurring. There was neural foraminal narrowing on the left at C4-5; and on the right at C5-6; and the left at C6-7. There was moderate spinal stenosis from C4-5 through C6-7.
On April 23, Dr. Park noted the employee's past history was significant for a 1987 neck injury and trouble with his right arm which recovered without much problem. In 1994, he had another neck injury, recovered uneventfully and was gainfully employed. On March 16, the employee fell at work and sustained neck pain with a new onset of left arm and back pain. He had altered sensation of the left C6 and C7 distribution and diminished strength in his biceps, triceps, and deltoids. A CT myelogram showed severe canal stenosis at C4-5, C5-6 and C6-7 with bilateral foraminal encroachment. The MRI showed spinal cord edema at C4-5 suggesting possible recent spinal cord injury. Dr. Park's impression was significant left-sided stenosis and bilateral foraminal stenosis with new symptoms of radiculopathy from the March 16, 2001 injury that involved the C6 and C7 nerve roots, and possibly the C5 nerve root. On May 3, Dr. Park performed a C4-5, C5-6, and C6-7 anterior cervical discectomy, foraminotomy and interbody fusion.
The employee saw Dr. Chaudhari on May 15, who noted that the employee sustained severe torsional trauma to his cervical and lumbar spinal axis with very intense pain in the C5-6 distribution of the left arm and significant low back pain. The employee had atrophy of the right supraspinatus and deltoid with muscle power fairly well preserved. Xanax was prescribed.
On May 31, Dr. Park noted that the left arm pain was gone. In June, Dr. Park ordered low back therapy and the employee had 21 physical therapy sessions beginning on July 9 and ending on September 5. On July 30, the employee had significant worsening of the lumbar pain. On August 30, Dr. Park stated he was at maximum medical improvement from the cervical spine. Dr. Park ordered a discogram at L2-3, L3-4, L4-5, and L5-S1. Since the employee had no significant history of back trouble prior to his injury, Dr. Park concluded that the employee's lumbar problems were a result of the March 2001 accident and injury.
On September 13, Dr. Park performed a L2-3, L3-4, L4-5 and L4-S1 discogram which showed identical pain at L5-S1, similar pain at L4-5, dissimilar pain at L3-4 and a negative injection at L2-3. The post discogram CT scan showed an annular tear and extravasation of contrast at L4-5; and small tears at L3-4 and L5-S1 could not be excluded. On September 17, Dr. Park stated that the employee's discogram showed significant findings with pain reproduction at L4-5 and L5-S1. At L3-4 the pain was somewhat unlikely. There were disc herniations at L3-4 and L4-5. On October 5, Dr. Park performed a L3-4, L4-5, and L5-S1 bilateral laminotomies and micro discectomies; and fusion with instrumentation.
2002:
On January 10, Dr. Park noted that the employee was having persistent back pain and occasional pain into the right buttock and right thigh. The employee ambulated with an antalgic gait on the right. On February 4, the employee had low back pain but no leg pain and numbness in his elbows down into his hands. He ambulated with a somewhat stooped gait, had weakness of the right tibialis anterior and extensor hallucis longus with deep tendon reflexes that were depressed at the Achilles' tendon. Dr. Park continued therapy and stated that the employee could go back to work at a sedentary job with no lifting over five pounds. On February 7, the employee was limping; was using a cane; had increased leg pain and a new onset of bilateral foot numbness and tingling. Dr. Park modified the therapy. On March 4, Dr. Park noted x-rays showed a solid lumbar fusion. He ordered work conditioning for four weeks. After that he would be at maximum medical improvement; and if he had additional problems the employee would need to see him under his health insurance. The employee had 24 physical therapy visits from January 14 through March 25 .
A functional capacity evaluation was performed on March 11. Deficits in the musculoskeletal evaluation included gait posture, flexibility, range of motion, strength, neurological, soft tissue assessment and Waddell's. Isometric strength testing revealed consistency of effort on ten out of ten tests. Functional testing revealed his present lifting in the light category of work. The employee demonstrated a tolerance of walking, stair climb, stooping, overhead reaching, and forward reaching on an occasional basis and sitting and standing on a frequent basis. The results of the evaluation indicated that the employee was currently unable to demonstrate the critical job demands of a pipe fitter. The functional capacity testing was partially completed due to self limiting behaviors.
On April 15, 2002, Dr. Park stated that the functional capacity evaluation was invalid due to scoring in the maximal range on the Waddell's testing for magnified behavior. Dr. Park noted that he would use empiric data to estimate the functional capacity. It was Dr. Park's opinion that the employee should be able to lift up to fifty pounds; and that a typical person with the surgical treatment that he had, should be able to do these kinds of activities with minimal difficulty. Dr. Park restricted the employee on the frequency of bending, twisting, stooping, climbing, pushing or pulling fifty pounds and reaching overhead to one third of the time. Dr. Park stated at the employee was at maximum medical improvement and was released from care. On April 29 Dr. Park estimated the employee's partial permanent impairment rating at 40 % overall with 20 % for
the cervical spine and 20\% for the lumbar spine. Dr. Park did not feel that the employee was permanently disabled.
On May 20, 2002, Dr. Chaudhari stated that the employee has intractable pain in the low back that radiated down into his left lower extremity. The employee was walking with a cane. Dr. Chaudhari diagnosed failed back syndrome with intractable pain.
The employee was seen by Dr. Kamath, a psychiatrist, on June 27 for depression. The 1987 traumatic accident was noted as was the March 2001 accident. Dr. Kamath stated that the employee was having chronic grief reaction due to loss of health, earning ability and self esteem. Dr. Kamath diagnosed mild depressive episode, major depression mild, single episode; prescribed Celexa, and recommended counseling. Dr. Kamath saw the employee in July, August and November. In November, Dr. Chaudhari stated that the employee had very minimal, if at all, benefit from the fusion. He had significant low back pain that radiated to the left lower extremity. Medications did not control the perpetual intractable pain.